Title : Sustained morphine treatment augments basal CGRP release from cultured primary sensory neurons in a Raf-1 dependent manner.

Pub. Date : 2008 Apr 28

PMID : 18328477






2 Functional Relationships(s)
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1 The selective PKA inhibitor, H-89, attenuates the sustained morphine-mediated augmentation of basal CGRP release, indicating that the cAMP/PKA pathway plays an important role in regulation of CGRP release from sensory neurons. N-(2-(4-bromocinnamylamino)ethyl)-5-isoquinolinesulfonamide calcitonin-related polypeptide alpha Rattus norvegicus
2 The selective PKA inhibitor, H-89, attenuates the sustained morphine-mediated augmentation of basal CGRP release, indicating that the cAMP/PKA pathway plays an important role in regulation of CGRP release from sensory neurons. N-(2-(4-bromocinnamylamino)ethyl)-5-isoquinolinesulfonamide calcitonin-related polypeptide alpha Rattus norvegicus