Title : Noradrenaline release by activation of κ-bungarotoxin-sensitive nicotinic acetylcholine receptors participates in long-term potentiation-like response induced by nicotine.

Pub. Date : 2007 Dec 25

PMID : 18157476






3 Functional Relationships(s)
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1 Our previous experiments showed that with the single pulses evoking 80% of the maximal population spike (PS) amplitude, nicotine (10 mumol/L) induced LTP-like response in the hippocampal CA1 region. Nicotine carbonic anhydrase 1 Homo sapiens
2 In the present study, the nicotinic acetylcholine receptor (nAChR) subtypes and relevant neurotransmitter releases involved in LTP-like response induced by nicotine were investigated by extracellularly recording the PS in the pyramidal cell layer in the hippocampal CA1 region in vitro. Nicotine carbonic anhydrase 1 Homo sapiens
3 The results suggest that noradrenaline release secondary to the activation of kappa-bungarotoxin-sensitive nAChRs participates in LTP-like response induced by nicotine in the hippocampal CA1 region. Nicotine carbonic anhydrase 1 Homo sapiens