Title : Aberrant V(D)J recombination is not required for rapid development of H2ax/p53-deficient thymic lymphomas with clonal translocations.

Pub. Date : 2008 Feb 15

PMID : 17855626






1 Functional Relationships(s)
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Compound Name
Protein Name
Organism
1 Thus, H2AX is essential for suppressing the transformation of developing thymocytes arising from the aberrant repair of spontaneous DSBs. dsbs H2A.X variant histone Mus musculus