Title : Involvement of c-Jun NH2-terminal kinase and nitric oxide-mediated mitochondria-dependent intrinsic pathway signaling in cardiotoxin-induced muscle cell death: role of testosterone.

Pub. Date : 2007 Nov

PMID : 17786558






1 Functional Relationships(s)
Download
Sentence
Compound Name
Protein Name
Organism
1 Ctx treatment also resulted in the inactivation of BCL-2 through phosphorylation at serine 70, thereby perturbing the BAX/BCL-2 rheostat, and the subsequent activation of the cytochrome c-mediated death pathway. Serine BCL2-associated X protein Mus musculus