Title : Bcr-Abl stabilizes beta-catenin in chronic myeloid leukemia through its tyrosine phosphorylation.

Pub. Date : 2007 Mar 7

PMID : 17318191






1 Functional Relationships(s)
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1 Self-renewal of Bcr-Abl(+) chronic myeloid leukemia (CML) cells is sustained by a nuclear activated serine/threonine-(S/T) unphosphorylated beta-catenin. Threonine ABL proto-oncogene 1, non-receptor tyrosine kinase Homo sapiens