Pub. Date : 2006 May
PMID : 16817862
4 Functional Relationships(s)Download |
Sentence | Compound Name | Protein Name | Organism |
| 1 | Using an in vitro oxygen-glucose deprivation (OGD) model of ischemia, we found that N-methyl-D-aspartate (NMDA) responses were enhanced in the more susceptible CA1 pyramidal cells and transiently depressed in the resistant CA3 pyramidal cells. | N-Methylaspartate | carbonic anhydrase 1 | Homo sapiens |
| 2 | Using an in vitro oxygen-glucose deprivation (OGD) model of ischemia, we found that N-methyl-D-aspartate (NMDA) responses were enhanced in the more susceptible CA1 pyramidal cells and transiently depressed in the resistant CA3 pyramidal cells. | N-Methylaspartate | carbonic anhydrase 1 | Homo sapiens |
| 3 | The long-lasting potentiation of NMDA responses in CA1 cells was associated with delayed cell death and was prevented by blocking tyrosine kinase-dependent up-regulation of NMDA receptor function. | N-Methylaspartate | carbonic anhydrase 1 | Homo sapiens |
| 4 | These results suggest that energy deprivation differentially shifts the intracellular equilibrium between the tyrosine kinase and phosphatase activities that modulate NMDA responses in CA1 and CA3 pyramidal cells. | N-Methylaspartate | carbonic anhydrase 1 | Homo sapiens |