Title : The dipeptide Arg-Gln inhibits retinal neovascularization in the mouse model of oxygen-induced retinopathy.

Pub. Date : 2006 Jul

PMID : 16799062






3 Functional Relationships(s)
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Compound Name
Protein Name
Organism
1 This study was conducted to examine the effect of the dipeptide arginyl-glutamine (Arg-Gln) on vascular endothelial cell growth factor (VEGF) levels in primary human retinal pigment epithelial (hRPE) cell cultures and on inhibition of neovascularization in the oxygen-induced retinopathy (OIR) model. Dipeptides vascular endothelial growth factor A Homo sapiens
2 Similarly the dipeptide reduced VEGF expression in hRPE cells, a cell type likely to respond to retinal hypoxia by expressing VEGF. Dipeptides vascular endothelial growth factor A Homo sapiens
3 Similarly the dipeptide reduced VEGF expression in hRPE cells, a cell type likely to respond to retinal hypoxia by expressing VEGF. Dipeptides vascular endothelial growth factor A Homo sapiens