Title : Excessive and precocious glutamate release in a mouse model of amyotrophic lateral sclerosis.

Pub. Date : 2004 May

PMID : 15033338






3 Functional Relationships(s)
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1 In SOD1(-)/G93A(-) or SOD1(+) mice glycine evoked [(3)H]d-ASP and [(3)H]GABA release, while GABA caused [3H]D-ASP, but not [3H]glycine, release. gamma-Aminobutyric Acid superoxide dismutase 1, soluble Mus musculus
2 The glycine-evoked release of [3H]D-ASP, but not that of [3H]GABA, and the GABA-evoked [3H]D-ASP release, but not that of [3H]glycine, were more pronounced in SOD1-G93A(+) than in SOD1(+) mice. gamma-Aminobutyric Acid superoxide dismutase 1, soluble Mus musculus
3 The glycine-evoked release of [3H]D-ASP, but not that of [3H]GABA, and the GABA-evoked [3H]D-ASP release, but not that of [3H]glycine, were more pronounced in SOD1-G93A(+) than in SOD1(+) mice. gamma-Aminobutyric Acid superoxide dismutase 1, soluble Mus musculus