Title : JNK activation is a mediator of arsenic trioxide-induced apoptosis in acute promyelocytic leukemia cells.

Pub. Date : 2004 May 1

PMID : 14701702






1 Functional Relationships(s)
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1 Furthermore, treatment of arsenic trioxide-sensitive APL cells with the JNK inhibitor, dicumarol, significantly increased growth and survival in response to As(2)O(3) but did not protect cells from doxorubicin. Dicumarol mitogen-activated protein kinase 8 Homo sapiens