Title : Selegiline reduces N-methyl-D-aspartic acid induced perturbation of neurotransmission but it leaves NMDA receptor dependent long-term potentiation intact in the hippocampus.

Pub. Date : 2003 Nov

PMID : 14628188






1 Functional Relationships(s)
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1 This study examined the effects of monoamine oxidase (MAO) inhibitors on N-methyl-D-aspartic acid (NMDA)-induced perturbation of neurotransmission and normal NMDA-receptor dependent function (long-term potentiation, LTP) in the CA1 field of hippocampus. N-Methylaspartate carbonic anhydrase 1 Homo sapiens