Title : Antisense oligonucleotide inhibition of Bcl-xL and Bid expression in liver regulates responses in a mouse model of Fas-induced fulminant hepatitis.

Pub. Date : 2003 Oct

PMID : 12893848






3 Functional Relationships(s)
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Protein Name
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1 Antisense oligonucleotide inhibition of Bcl-xL and Bid expression in liver regulates responses in a mouse model of Fas-induced fulminant hepatitis. ammonium ferrous sulfate BCL2-like 1 Mus musculus
2 Reduction of Bcl-xL expression resulted in a potentiation of Fas-mediated apoptosis in liver and significant increase of the lethality of Fas-mediated fulminant hepatitis (p < 0.0001). ammonium ferrous sulfate BCL2-like 1 Mus musculus
3 Reduction of Bcl-xL expression resulted in a potentiation of Fas-mediated apoptosis in liver and significant increase of the lethality of Fas-mediated fulminant hepatitis (p < 0.0001). ammonium ferrous sulfate BCL2-like 1 Mus musculus