Title : [Molecular mechanism of ALS and a possible gene therapy].

Pub. Date : 1999 Jan

PMID : 10377808






1 Functional Relationships(s)
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1 These results suggest that familial ALS with different mutations of the Cu/Zn SOD gene showed each clinical characteristics, that nitration of protein-tyrosine residue is upregulated in motor neurons of the spinal cord of ALS, and that there could be a possible future therapy of ALS with exogenous gene transfer. Tyrosine superoxide dismutase 1 Homo sapiens