Title : Protein-damaging stresses activate c-Jun N-terminal kinase via inhibition of its dephosphorylation: a novel pathway controlled by HSP72.

Pub. Date : 1999 Apr

PMID : 10082520






1 Functional Relationships(s)
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1 Conversely, exposure of cells to heat shock and other protein-damaging conditions, including ethanol, arsenite, and oxidative stress, strongly reduced the rate of JNK dephosphorylation. arsenite mitogen-activated protein kinase 8 Homo sapiens