27 Article(s)Download |
PMID | Title | Pub. Year | #Total Relationships |
1 | 35512188 | Lenalidomide promotes the development of TP53-mutated therapy-related myeloid neoplasms. | 2022 May 5 | 5 |
2 | 32251398 | Loss of lenalidomide-induced megakaryocytic differentiation leads to therapy resistance in del(5q) myelodysplastic syndrome. | 2020 May | 1 |
3 | 32323826 | Antitumor effect of lenalidomide in malignant glioma cell lines. | 2020 May | 1 |
4 | 30598450 | Dual inhibition of MDM2 and MDM4 in virus-positive Merkel cell carcinoma enhances the p53 response. | 2019 Jan 15 | 1 |
5 | 30608891 | Lenalidomide for the treatment of mantle cell lymphoma. | 2019 Apr | 1 |
6 | 29269520 | Emergence and evolution of TP53 mutations are key features of disease progression in myelodysplastic patients with lower-risk del(5q) treated with lenalidomide. | 2018 Apr | 1 |
7 | 30097406 | SOHO State of the Art and Next Questions: Management of Myelodysplastic Syndromes With Deletion 5q. | 2018 Oct | 1 |
8 | 27884971 | Progression in patients with low- and intermediate-1-risk del(5q) myelodysplastic syndromes is predicted by a limited subset of mutations. | 2017 Mar | 1 |
9 | 27967292 | The role of p53 in myelodysplastic syndromes and acute myeloid leukemia: molecular aspects and clinical implications. | 2017 Aug | 1 |
10 | 29296763 | A case of lenalidomide-dependent myelodysplastic syndrome. | 2017 Jul 11 | 1 |
11 | 26967821 | Lenalidomide treatment and prognostic markers in relapsed or refractory chronic lymphocytic leukemia: data from the prospective, multicenter phase-II CLL-009 trial. | 2016 Mar 11 | 1 |
12 | 27133825 | Prevalence, clonal dynamics and clinical impact of TP53 mutations in patients with myelodysplastic syndrome with isolated deletion (5q) treated with lenalidomide: results from a prospective multicenter study of the german MDS study group (GMDS). | 2016 Sep | 1 |
13 | 26075044 | Recent Advances in the 5q- Syndrome. | 2015 | 1 |
14 | 24627193 | Myelodysplastic syndromes with 5q deletion: pathophysiology and role of lenalidomide. | 2014 May | 1 |
15 | 24682512 | p53 protein expression independently predicts outcome in patients with lower-risk myelodysplastic syndromes with del(5q). | 2014 Jun | 1 |
16 | 24990888 | Lenalidomide inhibits the proliferation of CLL cells via a cereblon/p21(WAF1/Cip1)-dependent mechanism independent of functional p53. | 2014 Sep 4 | 2 |
17 | 25295231 | PP2A: The Achilles Heal in MDS with 5q Deletion. | 2014 | 1 |
18 | 22525275 | Lenalidomide promotes p53 degradation by inhibiting MDM2 auto-ubiquitination in myelodysplastic syndrome with chromosome 5q deletion. | 2013 Feb 28 | 3 |
19 | 23614682 | Response to lenalidomide in myelodysplastic syndromes with del(5q): influence of cytogenetics and mutations. | 2013 Jul | 2 |
20 | 24043769 | TP53 suppression promotes erythropoiesis in del(5q) MDS, suggesting a targeted therapeutic strategy in lenalidomide-resistant patients. | 2013 Oct 1 | 2 |
21 | 21968939 | Lenalidomide enhances the anti-prostate cancer activity of docetaxel in vitro and in vivo. | 2012 Jun 1 | 1 |
22 | 22109882 | The immunomodulatory drug lenalidomide restores a vitamin D sensitive phenotype to the vitamin D resistant breast cancer cell line MDA-MB-231 through inhibition of BCL-2: potential for breast cancer therapeutics. | 2012 Feb | 1 |
23 | 22261445 | p53 nuclear expression correlates with hemizygous TP53 deletion and predicts an adverse outcome for patients with relapsed/refractory multiple myeloma treated with lenalidomide. | 2012 Feb | 4 |
24 | 22933333 | Incidence of 17p deletions and TP53 mutation in myelodysplastic syndrome and acute myeloid leukemia with 5q deletion. | 2012 Dec | 1 |
25 | 23556117 | Efficacy and safety of lenalidomide in patients with myelodysplastic syndrome with chromosome 5q deletion. | 2012 Apr | 1 |
26 | 21321360 | Lenalidomide targets clonogenic side population in multiple myeloma: pathophysiologic and clinical implications. | 2011 Apr 28 | 1 |
27 | 19797731 | Clonal heterogeneity in the 5q- syndrome: p53 expressing progenitors prevail during lenalidomide treatment and expand at disease progression. | 2009 Dec | 1 |