8 Article(s)Download |
PMID | Title | Pub. Year | #Total Relationships |
1 | 27098371 | Altered development in GABA co-release shapes glycinergic synaptic currents in cultured spinal slices of the SOD1(G93A) mouse model of amyotrophic lateral sclerosis. | 2016 Jul 1 | 1 |
2 | 21414903 | Glycine receptor channels in spinal motoneurons are abnormal in a transgenic mouse model of amyotrophic lateral sclerosis. | 2011 Feb 23 | 1 |
3 | 17706944 | Antioxidant strategies based on tomato-enriched food or pyruvate do not affect disease onset and survival in an animal model of amyotrophic lateral sclerosis. | 2007 Sep 7 | 1 |
4 | 16309674 | Molecular and synaptic changes in the hippocampus underlying superior spatial abilities in pre-symptomatic G93A+/+ mice overexpressing the human Cu/Zn superoxide dismutase (Gly93 --> ALA) mutation. | 2006 Feb | 1 |
5 | 16436205 | Primary glia expressing the G93A-SOD1 mutation present a neuroinflammatory phenotype and provide a cellular system for studies of glial inflammation. | 2006 Jan 25 | 1 |
6 | 15885796 | Glutamate release induced by activation of glycine and GABA transporters in spinal cord is enhanced in a mouse model of amyotrophic lateral sclerosis. | 2005 Oct | 5 |
7 | 15033338 | Excessive and precocious glutamate release in a mouse model of amyotrophic lateral sclerosis. | 2004 May | 4 |
8 | 12684256 | Activation of a glycine transporter on spinal cord neurons causes enhanced glutamate release in a mouse model of amyotrophic lateral sclerosis. | 2003 Mar | 1 |