Methylphenidate

catechol-O-methyltransferase ; Homo sapiens







11 Article(s)
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1 35457347 Influence of COMT (rs4680) and DRD2 (rs1076560, rs1800497) Gene Polymorphisms on Safety and Efficacy of Methylphenidate Treatment in Children with Fetal Alcohol Spectrum Disorders. 2022 Apr 8 1
2 31164811 Heritable Differences in Catecholamine Signaling Modulate Susceptibility to Trauma and Response to Methylphenidate Treatment: Relevance for PTSD. 2019 3
3 27121430 The Effect of Single Dose Methylphenidate on Neurometabolites according to COMT Gene Val158Met Polymorphism in the Patient with Attention Deficit Hyperactivity Disorder: A Study Using Magnetic Resonance Spectroscopy. 2016 May 31 5
4 24035255 No association between catechol-O-methyltransferase (COMT) genotype and attention deficit hyperactivity disorder (ADHD) in Japanese children. 2014 Aug 1
5 24763183 Catechol-O-methyltransferase Val158-Met polymorphism and a response of hyperactive-impulsive symptoms to methylphenidate: A replication study from South Korea. 2014 Jul 3
6 23924290 Effect of COMT Val158Met genotype on attention and response to methylphenidate following traumatic brain injury. 2013 2
7 21550019 Catechol-O-methyltransferase valine158methionine polymorphism moderates methylphenidate effects on oppositional symptoms in boys with attention-deficit/hyperactivity disorder. 2011 Aug 1 1
8 18214865 Catechol-O-methyltransferase Val158Met polymorphism is associated with methylphenidate response in ADHD children. 2008 Dec 5 3
9 18580877 COMT Val108/158Met polymorphism and the modulation of task-oriented behavior in children with ADHD. 2008 Dec 1
10 18703939 Association of the catechol-O-methyltransferase polymorphism with methylphenidate response in a classroom setting in children with attention-deficit hyperactivity disorder. 2008 Sep 3
11 10490706 Haplotype relative risk study of catechol-O-methyltransferase (COMT) and attention deficit hyperactivity disorder (ADHD): association of the high-enzyme activity Val allele with ADHD impulsive-hyperactive phenotype. 1999 Oct 15 1