| 1 |
35368876 | Phenethyl Isothiocyanate Suppresses the Proinflammatory Cytokines in Human Glioblastoma Cells through the PI3K/Akt/NF-κB Signaling Pathway In Vitro. | 2022 |
2 |
| 2 |
32322335 | β-Phenethyl Isothiocyanate Induces Cell Death in Human Osteosarcoma through Altering Iron Metabolism, Disturbing the Redox Balance, and Activating the MAPK Signaling Pathway. | 2020 |
1 |
| 3 |
23788642 | The Ras GTPase-activating-like protein IQGAP1 mediates Nrf2 protein activation via the mitogen-activated protein kinase/extracellular signal-regulated kinase (ERK) kinase (MEK)-ERK pathway. | 2013 Aug 2 |
1 |
| 4 |
21595016 | Phenethyl isothiocyanate sensitizes human cervical cancer cells to apoptosis induced by cisplatin. | 2011 Oct |
1 |
| 5 |
21702500 | Effect of β-phenylethyl isothiocyanate from cruciferous vegetables on growth inhibition and apoptosis of cervical cancer cells through the induction of death receptors 4 and 5. | 2011 Aug 10 |
1 |
| 6 |
16624391 | Phenethyl isothiocyanate (PEITC) inhibits growth of ovarian cancer cells by inducing apoptosis: role of caspase and MAPK activation. | 2006 Oct |
1 |
| 7 |
16928811 | Mechanism of action of isothiocyanates: the induction of ARE-regulated genes is associated with activation of ERK and JNK and the phosphorylation and nuclear translocation of Nrf2. | 2006 Aug |
5 |
| 8 |
12819185 | The roles of JNK and apoptotic signaling pathways in PEITC-mediated responses in human HT-29 colon adenocarcinoma cells. | 2003 Aug |
2 |
| 9 |
12097262 | Phenethyl isothiocyanate-induced apoptosis in p53-deficient PC-3 human prostate cancer cell line is mediated by extracellular signal-regulated kinases. | 2002 Jul 1 |
2 |
| 10 |
11768769 | Induction of xenobiotic enzymes by the MAP kinase pathway and the antioxidant or electrophile response element (ARE/EpRE). | 2001 Aug-Nov |
1 |
| 11 |
12671299 | Differential activation of MAPK and ICE/Ced-3 protease in chemical-induced apoptosis. The role of oxidative stress in the regulation of mitogen-activated protein kinases (MAPKs) leading to gene expression and survival or activation of caspases leading to apoptosis. | 1998 Jun |
2 |