Title : Anergic T cells are defective in both jun NH2-terminal kinase and mitogen-activated protein kinase signaling pathways.

Pub. Date : 1996 May 1

PMID : 8642312






1 Functional Relationships(s)
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1 Furthermore, our data show that ERK and JNK activities are restored when anergy is induced in the presence of the protein synthesis inhibitor cycloheximide, or when anergic T cells are allowed to proliferate in response to exogenous IL-2. Cycloheximide mitogen-activated protein kinase 1 Mus musculus