Title : LARS2 Regulates Apoptosis via ROS-Mediated Mitochondrial Dysfunction and Endoplasmic Reticulum Stress in Ovarian Granulosa Cells.

Pub. Date : 2022

PMID : 35585880






3 Functional Relationships(s)
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1 LARS2 Regulates Apoptosis via ROS-Mediated Mitochondrial Dysfunction and Endoplasmic Reticulum Stress in Ovarian Granulosa Cells. Reactive Oxygen Species leucyl-tRNA synthetase 2, mitochondrial Homo sapiens
2 Meanwhile, LARS2 knockdown could induce mitochondrial dysfunction and accumulation of ROS levels. Reactive Oxygen Species leucyl-tRNA synthetase 2, mitochondrial Homo sapiens
3 Moreover, ROS was found to be involved in the antiproliferation, proapoptotic, and endoplasmic reticulum (ER) stress effects of LARS2 knockdown. Reactive Oxygen Species leucyl-tRNA synthetase 2, mitochondrial Homo sapiens