Title : GPR55 deficiency is associated with increased adiposity and impaired insulin signaling in peripheral metabolic tissues.

Pub. Date : 2019 Jan

PMID : 30148676






5 Functional Relationships(s)
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1 Emerging evidence indicates that G-protein coupled receptor 55 (GPR55), a nonclassic receptor of the endocannabinoid system that is activated by L-alpha-lysophosphatidylinositol and various cannabinoid ligands, may regulate endocrine function and energy metabolism. lysophosphatidylinositol G protein-coupled receptor 55 Mus musculus
2 Emerging evidence indicates that G-protein coupled receptor 55 (GPR55), a nonclassic receptor of the endocannabinoid system that is activated by L-alpha-lysophosphatidylinositol and various cannabinoid ligands, may regulate endocrine function and energy metabolism. lysophosphatidylinositol G protein-coupled receptor 55 Mus musculus
3 Emerging evidence indicates that G-protein coupled receptor 55 (GPR55), a nonclassic receptor of the endocannabinoid system that is activated by L-alpha-lysophosphatidylinositol and various cannabinoid ligands, may regulate endocrine function and energy metabolism. Cannabinoids G protein-coupled receptor 55 Mus musculus
4 Emerging evidence indicates that G-protein coupled receptor 55 (GPR55), a nonclassic receptor of the endocannabinoid system that is activated by L-alpha-lysophosphatidylinositol and various cannabinoid ligands, may regulate endocrine function and energy metabolism. Cannabinoids G protein-coupled receptor 55 Mus musculus
5 Sustained GPR55 antagonism in 3T3-L1 adipocytes enhanced expression of proteins implicated in lipogenesis and promoted triglyceride accumulation. Triglycerides G protein-coupled receptor 55 Mus musculus