Title : Recombinant club cell protein 16 (CC16) ameliorates cigarette smoke‑induced lung inflammation in a murine disease model of COPD.

Pub. Date : 2018 Aug

PMID : 29956762






2 Functional Relationships(s)
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1 Results indicated that rCC16 treatment ameliorated pathological damage in the lungs and reduced the production of tumor necrosis factor (TNF)-alpha, interleukin (IL)-6 and IL-8, which were induced by CS exposure. Cesium methylthioadenosine phosphorylase Rattus norvegicus
2 After rCC16 administration, endogenous CC16 was upregulated and the body weight of COPD mice was increased, whereas the opposite was observed in CS-exposed mice. Cesium methylthioadenosine phosphorylase Rattus norvegicus