Title : Leaf senescence induced by EGY1 defection was partially restored by glucose in Arabidopsis thaliana.

Pub. Date : 2015 Dec

PMID : 28510790






7 Functional Relationships(s)
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1 Leaf senescence induced by EGY1 defection was partially restored by glucose in Arabidopsis thaliana. Glucose Peptidase M50 family protein Arabidopsis thaliana
2 BACKGROUND: Ethylene-dependent gravitropism-deficient and yellow-green 1 (EGY1) protein is required for chloroplast development and photosynthesis conduction. ethylene Peptidase M50 family protein Arabidopsis thaliana
3 In accompany with this phenotype, leaf survival, chlorophyll content, Fv/Fm and soluble protein content decreased, and ion leakage increased significantly in egy1 mutants compared to WT plants. Chlorophyll Peptidase M50 family protein Arabidopsis thaliana
4 In addition, our results showed that the yellow-green phenotype, chlorophyll content and ion leakage of egy1 mutants was partially restored after exogenously applied glucose for 5 weeks. Glucose Peptidase M50 family protein Arabidopsis thaliana
5 At the same time, the expression of hexokinase 1 (HXK1) and/or senescence-associated gene 12 (SAG12) in egy1 mutants growing on 2 % glucose was lower than that in egy1 mutants without glucose. Glucose Peptidase M50 family protein Arabidopsis thaliana
6 At the same time, the expression of hexokinase 1 (HXK1) and/or senescence-associated gene 12 (SAG12) in egy1 mutants growing on 2 % glucose was lower than that in egy1 mutants without glucose. Glucose Peptidase M50 family protein Arabidopsis thaliana
7 CONCLUSION: EGY1-defection induced leaf senescence and this senescence was partially restored by glucose in A. thaliana. Glucose Peptidase M50 family protein Arabidopsis thaliana