Title : αE-catenin inhibits YAP/TAZ activity to regulate signalling centre formation during tooth development.

Pub. Date : 2016 Jul 13

PMID : 27405641






2 Functional Relationships(s)
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1 EK formation depends primarily on the signalling function of alphaE-catenin through YAP and its homologue TAZ, as opposed to its adhesive function, and combined deletion of Yap and Taz rescues the EK defects caused by loss of alphaE-catenin. H-Glu-Lys-OH tafazzin, phospholipid-lysophospholipid transacylase Mus musculus
2 EK formation depends primarily on the signalling function of alphaE-catenin through YAP and its homologue TAZ, as opposed to its adhesive function, and combined deletion of Yap and Taz rescues the EK defects caused by loss of alphaE-catenin. H-Glu-Lys-OH tafazzin, phospholipid-lysophospholipid transacylase Mus musculus