Title : Loss of central auditory processing in a mouse model of Canavan disease.

Pub. Date : 2014

PMID : 24826990






4 Functional Relationships(s)
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Sentence
Compound Name
Protein Name
Organism
1 ASPA-deficiency is characterized by severe psychomotor retardation, and excessive levels of the ASPA substrate N-acetylaspartate (NAA). N-acetylaspartate aspartoacylase Mus musculus
2 ASPA-deficiency is characterized by severe psychomotor retardation, and excessive levels of the ASPA substrate N-acetylaspartate (NAA). N-acetylaspartate aspartoacylase Mus musculus
3 ASPA-deficiency is characterized by severe psychomotor retardation, and excessive levels of the ASPA substrate N-acetylaspartate (NAA). N-acetylaspartate aspartoacylase Mus musculus
4 ASPA-deficiency is characterized by severe psychomotor retardation, and excessive levels of the ASPA substrate N-acetylaspartate (NAA). N-acetylaspartate aspartoacylase Mus musculus