Pub. Date : 1991 May-Jun
PMID : 1909512
4 Functional Relationships(s)Download |
Sentence | Compound Name | Protein Name | Organism |
| 1 | DMBA treatment induced binding of ras oncogene product ras p21 to [alpha 32P] GTP, altered estradiol metabolism by increasing the ratio of C16 alpha/C2 hydroxylation in favor of 16 alpha-OHE1 formation, and exhibited high frequency of MAL. | 6,11-dimethylbenzo(b)naphtho(2,3-d)thiophene | cyclin-dependent kinase inhibitor 1A (P21) | Mus musculus |
| 2 | DMBA treatment induced binding of ras oncogene product ras p21 to [alpha 32P] GTP, altered estradiol metabolism by increasing the ratio of C16 alpha/C2 hydroxylation in favor of 16 alpha-OHE1 formation, and exhibited high frequency of MAL. | [alpha 32p] gtp | cyclin-dependent kinase inhibitor 1A (P21) | Mus musculus |
| 3 | DMBA treatment induced binding of ras oncogene product ras p21 to [alpha 32P] GTP, altered estradiol metabolism by increasing the ratio of C16 alpha/C2 hydroxylation in favor of 16 alpha-OHE1 formation, and exhibited high frequency of MAL. | Estradiol | cyclin-dependent kinase inhibitor 1A (P21) | Mus musculus |
| 4 | DMBA treatment induced binding of ras oncogene product ras p21 to [alpha 32P] GTP, altered estradiol metabolism by increasing the ratio of C16 alpha/C2 hydroxylation in favor of 16 alpha-OHE1 formation, and exhibited high frequency of MAL. | -ohe1 | cyclin-dependent kinase inhibitor 1A (P21) | Mus musculus |