Title : Overexpression of catalase or Bcl-2 delays or prevents alterations in phospholipid metabolism during glucocorticoid-induced apoptosis in WEHI7.2 cells.

Pub. Date : 2003 Oct 21

PMID : 14572898






2 Functional Relationships(s)
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Compound Name
Protein Name
Organism
1 The steroid-resistant Hb12 cells showed a dramatic increase in glycerophosphocholine (GPC) content, suggesting increased phospholipid turnover may contribute to the anti-apoptotic mechanism of Bcl-2. Glycerylphosphorylcholine B cell leukemia/lymphoma 2 Mus musculus
2 The steroid-resistant Hb12 cells showed a dramatic increase in glycerophosphocholine (GPC) content, suggesting increased phospholipid turnover may contribute to the anti-apoptotic mechanism of Bcl-2. Glycerylphosphorylcholine B cell leukemia/lymphoma 2 Mus musculus