Title : Cell cycle arrest in ovarian cancer cell lines does not depend on p53 status upon treatment with cytostatic drugs.

Pub. Date : 1998 Nov

PMID : 9772293






3 Functional Relationships(s)
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Compound Name
Protein Name
Organism
1 The accumulated p53 was biochemically active, as measured in a transient transfection assay upon treatment with gemcitabine, cisplatin, etoposide, and Taxol. Paclitaxel tumor protein p53 Homo sapiens
2 Activity was dependent on the drug dose applied and proportional to the level of accumulated p53, except for Taxol-induced p53 accumulation which correlated inversely with p53 biochemical activity. Paclitaxel tumor protein p53 Homo sapiens
3 Activity was dependent on the drug dose applied and proportional to the level of accumulated p53, except for Taxol-induced p53 accumulation which correlated inversely with p53 biochemical activity. Paclitaxel tumor protein p53 Homo sapiens