Title : Inhibition of nitric oxide synthesis increases adenosine production via an extracellular pathway through activation of protein kinase C.

Pub. Date : 1997 Sep 2

PMID : 9315551






2 Functional Relationships(s)
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1 The intracoronary administration of alpha,beta-methyleneadenosine 5"-diphosphate, an inhibitor of ecto-5"-nucleotidase, or GF109203X or calphostin C, both of which are PKC inhibitors, attenuated the L-NAME-induced increases in adenosine levels and ecto-5"-nucleotidase activity. alpha,beta-methyleneadenosine 5'-diphosphate 5'-nucleotidase ecto Homo sapiens
2 The intracoronary administration of alpha,beta-methyleneadenosine 5"-diphosphate, an inhibitor of ecto-5"-nucleotidase, or GF109203X or calphostin C, both of which are PKC inhibitors, attenuated the L-NAME-induced increases in adenosine levels and ecto-5"-nucleotidase activity. alpha,beta-methyleneadenosine 5'-diphosphate 5'-nucleotidase ecto Homo sapiens