Title : Forskolin delays the ethanol-induced desensitization of hypothalamic beta-endorphin neurons in primary cultures.

Pub. Date : 1997 May

PMID : 9161608






6 Functional Relationships(s)
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1 Ethanol and its metabolite acetaldehyde have been shown to stimulate immunoreactive beta-endorphin (IR-beta-EP) secretion from hypothalamic neurons in primary cultures. Acetaldehyde proopiomelanocortin Homo sapiens
2 Ethanol and its metabolite acetaldehyde have been shown to stimulate immunoreactive beta-endorphin (IR-beta-EP) secretion from hypothalamic neurons in primary cultures. Acetaldehyde proopiomelanocortin Homo sapiens
3 Acetaldehyde stimulated IR-beta-EP secretion from this culture for a period of 48 hr, but the IR-beta-EP secretory response to acetaldehyde reduced gradually with time during the first 48-hr period and reached the basal level at 72 hr. Acetaldehyde proopiomelanocortin Homo sapiens
4 Acetaldehyde stimulated IR-beta-EP secretion from this culture for a period of 48 hr, but the IR-beta-EP secretory response to acetaldehyde reduced gradually with time during the first 48-hr period and reached the basal level at 72 hr. Acetaldehyde proopiomelanocortin Homo sapiens
5 However, reduced IR- beta-EP secretory response to acetaldehyde with time was associated with the time-dependent increase in cell death. Acetaldehyde proopiomelanocortin Homo sapiens
6 These results suggest that (i) chronic treatment with ethanol desensitizes beta-EP-secreting neurons due to reduced cellular functions and (ii) chronic acetaldehyde reduces beta-EP neurotransmission due to cell death. Acetaldehyde proopiomelanocortin Homo sapiens