Title : Deletion analysis of the dystrophin-actin binding domain.

Pub. Date : 1994 May 16

PMID : 8187894






1 Functional Relationships(s)
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1 Instead, our data suggest that sequences in the N-terminal 90 amino acids of dystrophin, excluding a conserved KTFT motif, contain the major site for interaction with actin. ktft dystrophin Homo sapiens