Title : The myasthenic (Eaton-Lambert) syndrome associated with carcinoma. Enzyme induction as a possible mechanism of paraneoplastic syndromes.

Pub. Date : 1982 Jul

PMID : 6287570






1 Functional Relationships(s)
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1 It is suggested that all previously described neuromuscular responses are compatible with new model: the subsynaptic apparatus produces excess acetylcholinesterase (AChE) which limits the endplate conductance changes produced by normal output of ACh. Acetylcholine acetylcholinesterase (Cartwright blood group) Homo sapiens