Title : Inhibition of Gli1 suppressed hyperglycemia-induced meibomian gland dysfunction by promoting pparĪ³ expression.

Pub. Date : 2022 Jul

PMID : 35594713






2 Functional Relationships(s)
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1 We further demonstrated that advanced glycation end products (AGEs) treatment also promoted the expression of Gli1 and ppargamma in organotypic cultured mouse MG. AGEs inhibitor Aminoguanidine suppressed high glucose caused Gli1 upregulation in organotypic cultured mouse MG. pimagedine GLI-Kruppel family member GLI1 Mus musculus
2 We further demonstrated that advanced glycation end products (AGEs) treatment also promoted the expression of Gli1 and ppargamma in organotypic cultured mouse MG. AGEs inhibitor Aminoguanidine suppressed high glucose caused Gli1 upregulation in organotypic cultured mouse MG. pimagedine GLI-Kruppel family member GLI1 Mus musculus