Title : Impaired signaling for neuromuscular synaptic maintenance is a feature of Motor Neuron Disease.

Pub. Date : 2022 Apr 25

PMID : 35468848






2 Functional Relationships(s)
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Protein Name
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1 Maintenance of the NMJ relies upon neural agrin (n-agrin) which, when released from the nerve terminal, activates the postsynaptic Muscle Specific Kinase (MuSK) signaling complex to stabilize clusters of acetylcholine receptors. Acetylcholine muscle associated receptor tyrosine kinase Homo sapiens
2 Maintenance of the NMJ relies upon neural agrin (n-agrin) which, when released from the nerve terminal, activates the postsynaptic Muscle Specific Kinase (MuSK) signaling complex to stabilize clusters of acetylcholine receptors. Acetylcholine muscle associated receptor tyrosine kinase Homo sapiens