Title : SNHG14 Upregulation Was a Molecular Mechanism Underlying MPP+ Neurotoxicity in Dopaminergic SK-N-SH Cells via SNHG14-miR-519a-3p-ATG10 ceRNA Pathway.

Pub. Date : 2022 Apr

PMID : 35349097






4 Functional Relationships(s)
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1 SNHG14 Upregulation Was a Molecular Mechanism Underlying MPP+ Neurotoxicity in Dopaminergic SK-N-SH Cells via SNHG14-miR-519a-3p-ATG10 ceRNA Pathway. mangion-purified polysaccharide (Candida albicans) small nucleolar RNA host gene 14 Homo sapiens
2 SNHG14 Upregulation Was a Molecular Mechanism Underlying MPP+ Neurotoxicity in Dopaminergic SK-N-SH Cells via SNHG14-miR-519a-3p-ATG10 ceRNA Pathway. mangion-purified polysaccharide (Candida albicans) small nucleolar RNA host gene 14 Homo sapiens
3 SNHG14 and ATG10 expression was increased, and miR-519a-3p was decreased in MPP+-treated SK-N-SH cells, and SNHG14 knockdown alleviated MPP+-induced SK-N-SH cell damage by regulating cell viability, cell cycle arrest, apoptosis, and oxidative stress. mangion-purified polysaccharide (Candida albicans) small nucleolar RNA host gene 14 Homo sapiens
4 Target silencing SNHG14 and restoring miR-519a-3p could prevent DAn from MPP+ toxicity via regulation of ATG10. mangion-purified polysaccharide (Candida albicans) small nucleolar RNA host gene 14 Homo sapiens