Title : Chronic androgen excess in female mice does not impact luteinizing hormone pulse frequency or putative GABAergic inputs to GnRH neurons.

Pub. Date : 2022 Apr

PMID : 35267218






3 Functional Relationships(s)
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1 Acute prenatal DHT exposure can also model the hyperandrogenism of PCOS, and this is accompanied by increased LH pulse frequency and increased GABAergic innervation of gonadotrophin-releasing hormone (GnRH) neurons. Dihydrotestosterone gonadotropin releasing hormone 1 Mus musculus
2 Acute prenatal DHT exposure can also model the hyperandrogenism of PCOS, and this is accompanied by increased LH pulse frequency and increased GABAergic innervation of gonadotrophin-releasing hormone (GnRH) neurons. Dihydrotestosterone gonadotropin releasing hormone 1 Mus musculus
3 We aimed to determine the impact of chronic exposure of female mice to DHT, which models the hyperandrogenism of PCOS, on pulsatile LH secretion and putative GABAergic input to GnRH neurons. Dihydrotestosterone gonadotropin releasing hormone 1 Mus musculus