Title : Aldolase A and Phospholipase D1 Synergistically Resist Alkylating Agents and Radiation in Lung Cancer.

Pub. Date : 2021

PMID : 35127525






4 Functional Relationships(s)
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1 Using the existing metabolomics platform, we demonstrated that lysophosphatidylethanolamine (LPE) and lysophosphatidylcholine (LPC) are the most critical metabolites, and are highly dependent on aldolase A (ALDOA). lysophosphatidylethanolamine aldolase, fructose-bisphosphate A Homo sapiens
2 Using the existing metabolomics platform, we demonstrated that lysophosphatidylethanolamine (LPE) and lysophosphatidylcholine (LPC) are the most critical metabolites, and are highly dependent on aldolase A (ALDOA). lysophosphatidylethanolamine aldolase, fructose-bisphosphate A Homo sapiens
3 Using the existing metabolomics platform, we demonstrated that lysophosphatidylethanolamine (LPE) and lysophosphatidylcholine (LPC) are the most critical metabolites, and are highly dependent on aldolase A (ALDOA). lysophosphatidylethanolamine aldolase, fructose-bisphosphate A Homo sapiens
4 Using the existing metabolomics platform, we demonstrated that lysophosphatidylethanolamine (LPE) and lysophosphatidylcholine (LPC) are the most critical metabolites, and are highly dependent on aldolase A (ALDOA). lysophosphatidylethanolamine aldolase, fructose-bisphosphate A Homo sapiens