Title : Cortical and Subcortical Dysmetabolism Are Dynamic Markers of Clinical Disability and Course in Anti-LGI1 Encephalitis.

Pub. Date : 2022 Mar

PMID : 35091466






4 Functional Relationships(s)
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1 BACKGROUND AND OBJECTIVES: This (18F)fluorodeoxyglucose (FDG) PET study evaluates the accuracy of semiquantitative measurement of putaminal hypermetabolism in identifying anti-leucine-rich, glioma-inactivated-1 (LGI1) protein autoimmune encephalitis (AE). Fluorodeoxyglucose F18 leucine rich glioma inactivated 1 Homo sapiens
2 BACKGROUND AND OBJECTIVES: This (18F)fluorodeoxyglucose (FDG) PET study evaluates the accuracy of semiquantitative measurement of putaminal hypermetabolism in identifying anti-leucine-rich, glioma-inactivated-1 (LGI1) protein autoimmune encephalitis (AE). Fluorodeoxyglucose F18 leucine rich glioma inactivated 1 Homo sapiens
3 BACKGROUND AND OBJECTIVES: This (18F)fluorodeoxyglucose (FDG) PET study evaluates the accuracy of semiquantitative measurement of putaminal hypermetabolism in identifying anti-leucine-rich, glioma-inactivated-1 (LGI1) protein autoimmune encephalitis (AE). Fluorodeoxyglucose F18 leucine rich glioma inactivated 1 Homo sapiens
4 BACKGROUND AND OBJECTIVES: This (18F)fluorodeoxyglucose (FDG) PET study evaluates the accuracy of semiquantitative measurement of putaminal hypermetabolism in identifying anti-leucine-rich, glioma-inactivated-1 (LGI1) protein autoimmune encephalitis (AE). Fluorodeoxyglucose F18 leucine rich glioma inactivated 1 Homo sapiens