Title : Targeting INMT and interrupting its methylation pathway for the treatment of castration resistant prostate cancer.

Pub. Date : 2021 Sep 29

PMID : 34587977






1 Functional Relationships(s)
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1 Importantly, INMT knockdown significantly increased the anticancer effect of the exogenous selenium compounds methaneseleninic acid (MSA) and Se-(Methyl)selenocysteine hydrochloride (MSC) as well as the endogenous metabolite Bis(7)-tacrine. msc indolethylamine N-methyltransferase Mus musculus