Title : The Chk2-PKM2 axis promotes metabolic control of vasculogenic mimicry formation in p53-mutated triple-negative breast cancer.

Pub. Date : 2021 Aug

PMID : 34244606






1 Functional Relationships(s)
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1 Overall, this study proposes a novel strategy of VM suppression through Chk2 induction, which prevents PKM2-mediated glucose flux in p53-mutated TNBC. Glucose tumor protein p53 Homo sapiens