Title : Nrf2 Activation Attenuates Acrylamide-Induced Neuropathy in Mice.

Pub. Date : 2021 Jun 1

PMID : 34206048






4 Functional Relationships(s)
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1 Moreover, co-administration of sulforaphane enhanced acrylamide-induced mRNA upregulation of Nrf2 and its downstream antioxidant proteins and suppressed acrylamide-induced mRNA upregulation of tumor necrosis factor alpha (TNF-alpha) and inducible nitric oxide synthase (iNOS) in the cerebral cortex. Acrylamide nitric oxide synthase 2, inducible Mus musculus
2 Moreover, co-administration of sulforaphane enhanced acrylamide-induced mRNA upregulation of Nrf2 and its downstream antioxidant proteins and suppressed acrylamide-induced mRNA upregulation of tumor necrosis factor alpha (TNF-alpha) and inducible nitric oxide synthase (iNOS) in the cerebral cortex. Acrylamide nitric oxide synthase 2, inducible Mus musculus
3 Moreover, co-administration of sulforaphane enhanced acrylamide-induced mRNA upregulation of Nrf2 and its downstream antioxidant proteins and suppressed acrylamide-induced mRNA upregulation of tumor necrosis factor alpha (TNF-alpha) and inducible nitric oxide synthase (iNOS) in the cerebral cortex. Acrylamide nitric oxide synthase 2, inducible Mus musculus
4 Moreover, co-administration of sulforaphane enhanced acrylamide-induced mRNA upregulation of Nrf2 and its downstream antioxidant proteins and suppressed acrylamide-induced mRNA upregulation of tumor necrosis factor alpha (TNF-alpha) and inducible nitric oxide synthase (iNOS) in the cerebral cortex. Acrylamide nitric oxide synthase 2, inducible Mus musculus