Title : Regulation of TRPML1 channel activity and inflammatory exosome release by endogenously produced reactive oxygen species in mouse podocytes.

Pub. Date : 2021 Jul

PMID : 34030116






2 Functional Relationships(s)
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1 Regulation of TRPML1 channel activity and inflammatory exosome release by endogenously produced reactive oxygen species in mouse podocytes. Reactive Oxygen Species mucolipin 1 Mus musculus
2 Based on these results, we conclude that endogenously produced ROS importantly contributes to inflammatory exosome secretion from podocytes through inhibition of TRPML1 channel activity, which may contribute to the initiation of glomerular inflammation during hHcy. Reactive Oxygen Species mucolipin 1 Mus musculus