Title : Understanding potential mechanisms of harm: the drivers of electronic cigarette-induced changes in alveolar macrophages, neutrophils, and lung epithelial cells.

Pub. Date : 2021 Aug 1

PMID : 34009037

1 Functional Relationships(s)
Compound Name
Protein Name
1 These effects are largely driven by free radical exposure, changes in PI3K/Akt signalling pathways, nicotine-induced reduction in phagocytosis receptors and impaired lipid homeostasis leading to a foam-like lipid laden phenotype. Nicotine AKT serine/threonine kinase 1 Homo sapiens