Title : Hydrogen sulfide ameliorates high glucose-induced pro-inflammation factors in HT-22 cells: Involvement of SIRT1-mTOR/NF-κB signaling pathway.

Pub. Date : 2021 Jun

PMID : 33765609






2 Functional Relationships(s)
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1 The administration of sodium hydrosulfide (NaHS, a H2S donor) or S-adenosylmethionine (SAMe, an allosteric activator of CBS) restored high glucose-induced downregulation of CBS and H2S levels. S-Adenosylmethionine cystathionine beta-synthase Mus musculus
2 The administration of sodium hydrosulfide (NaHS, a H2S donor) or S-adenosylmethionine (SAMe, an allosteric activator of CBS) restored high glucose-induced downregulation of CBS and H2S levels. S-Adenosylmethionine cystathionine beta-synthase Mus musculus