Title : Post-treatment hypermutation in a recurrent diffuse glioma with H3.3 p.G34 Mutation.

Pub. Date : 2021 Apr

PMID : 33296093






5 Functional Relationships(s)
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1 Post-treatment TMZ-associated hypermutation is a mechanism for TMZ resistance in recurrent isocitrate dehydrogenase (IDH) wildtype glioblastoma and may be seen in association with tumour progression in lower-grade IDH-mutant diffuse astrocytic gliomas. Temozolomide isocitrate dehydrogenase (NADP(+)) 1 Homo sapiens
2 Post-treatment TMZ-associated hypermutation is a mechanism for TMZ resistance in recurrent isocitrate dehydrogenase (IDH) wildtype glioblastoma and may be seen in association with tumour progression in lower-grade IDH-mutant diffuse astrocytic gliomas. Temozolomide isocitrate dehydrogenase (NADP(+)) 1 Homo sapiens
3 Post-treatment TMZ-associated hypermutation is a mechanism for TMZ resistance in recurrent isocitrate dehydrogenase (IDH) wildtype glioblastoma and may be seen in association with tumour progression in lower-grade IDH-mutant diffuse astrocytic gliomas. Temozolomide isocitrate dehydrogenase (NADP(+)) 1 Homo sapiens
4 Post-treatment TMZ-associated hypermutation is a mechanism for TMZ resistance in recurrent isocitrate dehydrogenase (IDH) wildtype glioblastoma and may be seen in association with tumour progression in lower-grade IDH-mutant diffuse astrocytic gliomas. Temozolomide isocitrate dehydrogenase (NADP(+)) 1 Homo sapiens
5 Post-treatment TMZ-associated hypermutation is a mechanism for TMZ resistance in recurrent isocitrate dehydrogenase (IDH) wildtype glioblastoma and may be seen in association with tumour progression in lower-grade IDH-mutant diffuse astrocytic gliomas. Temozolomide isocitrate dehydrogenase (NADP(+)) 1 Homo sapiens