Title : Regulation of Thiamine (Vitamin B1)-Dependent Metabolism in Mammals by p53.

Pub. Date : 2020 Jul

PMID : 33040724






1 Functional Relationships(s)
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1 Simultaneously, the activities of NAD+-dependent IDH, mitochondrial aspartate aminotransferase, and two malate dehydrogenase isoenzymes, whose genes were not predicted to have the p53-binding sequences near the transcription starting points, were upregulated by cisplatin. NAD tumor protein p53 Homo sapiens