Title : Exposure to hyperandrogen drives ovarian dysfunction and fibrosis by activating the NLRP3 inflammasome in mice.

Pub. Date : 2020 Nov 25

PMID : 32758727






2 Functional Relationships(s)
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1 After inhibiting NLRP3, dihydrotestosterone (DHT)-treated GCs demonstrated markedly decreased NLRP3, the inflammasome adapter protein ASC, C-terminal fragment of gasdermin D (GSDMD-C), AR and Cyp19alpha1 at the protein level. Dihydrotestosterone androgen receptor Mus musculus
2 After inhibiting NLRP3, dihydrotestosterone (DHT)-treated GCs demonstrated markedly decreased NLRP3, the inflammasome adapter protein ASC, C-terminal fragment of gasdermin D (GSDMD-C), AR and Cyp19alpha1 at the protein level. Dihydrotestosterone androgen receptor Mus musculus