Title : Activation of CB1R Promotes Lipopolysaccharide-Induced IL-10 Secretion by Monocytic Myeloid-Derived Suppressive Cells and Reduces Acute Inflammation and Organ Injury.

Pub. Date : 2020 Jun 15

PMID : 32385136






1 Functional Relationships(s)
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1 The anti-inflammatory effects of Delta9-THC in endotoxemic mice were reversed by a cannabinoid receptor type 1 (CB1R) inverse agonist (SR141716), and by clodronate-induced myeloid-cell depletion, but not by genetic invalidation or blockade of other putative Delta9-THC receptors, including cannabinoid receptor type 2, TRPV1, GPR18, GPR55, and GPR119. Dronabinol transient receptor potential cation channel, subfamily V, member 1 Mus musculus