Title : Activation of AMP-activated protein kinase during sepsis/inflammation improves survival by preserving cellular metabolic fitness.

Pub. Date : 2020 May

PMID : 32246808






2 Functional Relationships(s)
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1 Pharmacologic activation of AMPK with 5-aminoimidazole-4-carboxamide ribonucleotide (AICAR) or metformin during sepsis improved the survival, while AMPK inhibition with Compound C increased mortality, impaired mitochondrial respiration, decreased OCR, and disrupted TEC metabolic fitness. AICA ribonucleotide protein kinase AMP-activated non-catalytic subunit beta 1 Homo sapiens
2 Pharmacologic activation of AMPK with 5-aminoimidazole-4-carboxamide ribonucleotide (AICAR) or metformin during sepsis improved the survival, while AMPK inhibition with Compound C increased mortality, impaired mitochondrial respiration, decreased OCR, and disrupted TEC metabolic fitness. AICA ribonucleotide protein kinase AMP-activated non-catalytic subunit beta 1 Homo sapiens