Title : Abnormal Upregulation of GPR17 Receptor Contributes to Oligodendrocyte Dysfunction in SOD1 G93A Mice.

Pub. Date : 2020 Mar 31

PMID : 32244295






1 Functional Relationships(s)
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1 Moreover, OPCs isolated from spinal cord of SOD1G93A mice display defective differentiation compared to control cells, which is rescued by treatment with the GPR17 antagonist montelukast. montelukast G protein-coupled receptor 17 Mus musculus