Title : The Reduced Oligomerization of MAVS Mediated by ROS Enhances the Cellular Radioresistance.

Pub. Date : 2020

PMID : 32190169






1 Functional Relationships(s)
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1 Our current study demonstrated that knockdown of MAVS alleviated the radiation-induced mitochondrial dysfunction (mitochondrial membrane potential disruption and ATP production), downregulated the expressions of proapoptotic proteins, and reduced the generation of ROS in cells after irradiation. Adenosine Triphosphate mitochondrial antiviral signaling protein Homo sapiens